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- ischemic brain infarction apoplexy (IBIA) 缺血性中风
- ischemic brain infarction 缺血性脑梗塞
- The mechanisms contributed to ischemic brain injury involve excitotoxicity,calcium overload,free radical reaction,depolarization surrounding infarct,inflammation and apoptosis. 兴奋毒性、钙超载、自由基反应、梗死周围除极、炎症和凋亡是造成缺血性脑损伤的中心环节。
- Conclusion DWI are sensitivity in diagnosing super-acute cerebral ischemia and if combined with T2WI and FLAIR, it can distinguish the various stages of brain infarction. 结论DWI对诊断超急性脑梗死十分歌感,结合其他序列能对脑梗死做出明确分期。
- Conclusion:Tong Nao Ling has marked effect on brain infarction. 通脑灵治疗脑梗塞有较好疗效。
- Conclusion FG can reduce ischemic brain damage and protect cerebral tissue. 结论甘草总黄酮能减轻缺血性脑损害,保护脑组织。
- It has been reported that MMP-2 and MMP-9 are activated after onset of stroke in clinical practice as well as in experimental brain ischemia and inhibiting the activityof MMP-9 can reduce brain infarct. 临床上已有报道MMP-2及MMP-9的活性在脑中风发作后明显升高。 动物实验也显示局部脑缺血后MMP-9在早期就被激活,抑制MMP-9的活性可以减小脑梗塞面积。
- AIM: To investigate the mechanism for electroacupuncture in improving the plasticity of synapse after ischemic brain injury. 目的:探讨针刺对缺血性脑损伤后的突触可塑性促进作用的机制。
- Cerebral ischemia may induce upregulation of ANG and GFAP expression in the ischemic brain areas. 脑缺血可诱导缺血脑区ANG和GFAP的表达上调。
- Objective It is to discuss the clinical curative effect of fleabane injection on acute brain infarction. 目的探讨灯盏细辛注射液治疗急性脑梗死的临床疗效。
- To study the effects of glutathione on hypoxic ischemic brain damage in neonatal rats hippocampal. 研究谷胱甘肽对缺氧缺血脑损伤引起的神经细胞凋亡的作用。
- Objective: To observe the therapeutic effect of cinepazide maleate on brain infarct. 目的:观察桂哌齐特对脑梗死的治疗效果。
- Gabryel B,Trzeciak HI.Role of astrocytes in pathogenesis of ischemic brain injury[J].Neurotox Res,2001 ;3(2):205-21. 周琴;廖维靖;杨万同;等.;阿魏酸钠促进局灶性脑缺血再灌注后神经功能恢复和血管生成作用的研究[J]
- Methods The clinical datum of middle and young people's brain infarction is reviewed to analyse in 36 patients. 方法回顾分析36例中青年脑梗死的临床资料。
- Study on the correlation between CGG triplet repeat ploymorphism of VLDL-R gene and atherosclerosis brain infarction. 极低密度脂蛋白受体基因多态性与动脉粥样硬化性脑梗死的相关性研究。
- Cerebral ischemia activates endogenous angiogenic factors,and increase microvessel density and form new capillaries in ischemic brain tissues. 脑缺血能激活内源性血管生成因子,使缺血脑组织血管密度增高,新生毛细血管形成。
- Objective To evaluate the values of visual retention test (VRT) in the patients with silent brain infarction (SBI). 目的 评估视觉保持测验 (VRT)在无症状性脑梗死 (SBI)中的应用价值。
- On the other hand, cycloheximide could reduce the expression of P 53 and P 21 proteins in ischemic brain tissue also protect CA1 region from DND. 另外,放线菌酮可抑制脑组织P53、P21蛋白的表达,并对DND具有一定的保护作用。
- AIM: To evaluate the instant effect of naloxone for brain infarction with somatosensory evoked potentials (SEP). 目的 :应用躯体感觉诱发电位 (SEP)的脑功能评价法观察纳洛酮对脑梗死即刻效应。
- Objective To study the effect and possible mechanism of He Ne laser acupoint irradiation treatment on hypoxic ischemic brain damage (HIBD). 目的探讨氦氖激光穴位照射对新生鼠缺血缺氧性脑损伤的作用及其可能机制。