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- Background: Hepatic ischemia reperfusion (HIR) injury is a common acute disease of surgery. 肝脏缺血再灌注损伤(Hepatic ischemia reperfusion injury,HIR)是指肝脏缺血后重获血流灌注或氧供后,肝脏损伤会在缺血缺氧损伤的基础上进一步加重。
- Protective effect of heme oxygenase on hepatic ischemia reperfusion injury in rats FENG Zhi-jie, CHEN Xiang-ping. 血红素氧合酶;一氧化碳;肝;缺血再灌注损伤;氧化性应激
- Objective To study the effects of heat shock proteins (HSPs) in the course of hepatic ischemia reperfusion injury (HIRI), and analyze its mechanism. 目的了解热休克蛋白在肝缺血再灌注损伤过程中所处的位置、作用及其相关机理。
- Hepatic ischemia reperfusion leads to oxygen free radical producing excessively, calcium overload and ATP depletion, which destroy bile canalicular F-actin directly or indirectly. 4. 3.肝脏缺血再灌注造成氧自由基过量生成,细胞钙超载,ATP耗竭,直接或间接破坏胆小管F-actin微丝。
- hepatic ischemia reperfusion injury 肝缺血再灌注损伤
- Hepatic ischemia reperfusion injure 肝缺血再灌注损伤
- hepatic ischemic reperfusion injury 肝脏缺血再灌注损伤
- Experimental Studies on Protective Effect of Intra Oxygen Injection Against Hepatic Ischemia Reperfusion Injury 注射用内给氧对兔肝缺血再灌注损伤保护作用的实验研究
- hepatic ischemia reperfusion 肝脏缺血再灌注
- Objective:To evaluate the role of oxygen free radicals in hepatic ischemia and reperfusion injury(HIRI) and the influence of tiopronin. 目的:探讨氧自由基在肝缺血再灌注损伤(HIRI)中的作用及凯西莱的防护作用。
- Objective: To study the effects of the preconditioning with enflurane and isoflurane on hepatic ischemia and reperfusion injury. 目的: 研究安氟醚和异氟醚预处理对肝脏缺血/再灌注损害的影响。
- Study on the Effect of Hepatic Glycogen Content on Liver Ischemia Reperfusion Injury and Its Mechanism in Rabbits. 肝细胞糖原拮抗肝脏热缺血再灌注损伤相关机理的研究。
- Method: Hepatic ischemia and reperfusion medel in 60 SD rats was made by hepatic veins and artery reperfusion for 60 minutes following hepatic portal vein and artery occlusion for 30 minutes. 方法:建立SD大鼠肝脏缺血再灌注模型,第一肝门持续阻断30min后开放60min,60只大鼠随机分为6组,每组10只:A组(正常对照组),无肝脏缺血再灌注; B组(缺血再灌注组);
- After hepatic ischemia for 15 minutes and after reperfusion for 1 hour, 3 hours and 24 hours respectively, the hepatic tissue were resected for determining the activity of ATPase and for enzyme-histochemistry dyeing. 肝脏缺血15min,分别于再灌注后1、3、24h取肝组织测定Na+-K+-ATP酶、Ca2+-Mg2+-ATP酶水平,并行酶组织化学染色,观察Ca2+-ATP酶的活性变化。
- CONCLUSION DK can protect myocardium against ischemia reperfusion injury. 结论dk对大鼠心肌缺血再灌注损伤有保护作用。
- Hepatic ischemia and reperfusion injury 肝脏缺血再灌注损伤
- NGF could ease the delayed neuron death (DND) after ischemia reperfusion. NGF可以减轻海马迟发性神经细胞死亡 (DND)性损伤。
- Conclusion: Ischemia reperfusion could induce the apoptosis of the cell. 说明缺血再灌注损伤能诱发细胞凋亡。
- Objective To investigate the protective mechanism of flunarizine in gerbil brain after ischemia reperfusion. 目的进一步探讨盐酸氟桂嗪对缺血再灌注后神经细胞保护作用的机制。
- Objective:Study the effect of acusector on serum CK and LDH of cerebral ischemia reperfusion injury model in rats. 目的:研究电针对实验性脑缺血再灌注损伤大鼠血清中肌酸激酶(CK)和乳酸脱氢酶(LDH)的影响。