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drug resistance reversal fold 耐药逆转倍数
drug resistance reversal 耐药性逆转
Kirby-Bauer was used to detect the drug resistance. 采用kirby -Bauer法检测其耐药性。
K562/ADM had no drug resistance to As2O3 and matrine. 非细胞毒性剂量的三氧化二砷和苦参碱均可部分逆转有多药耐药表型的细胞株K562/ADM对阿霉素的耐药J险，二者联合应用效果优于单独应用，具有协同作用。
Such misuse of medicines can lead to drug resistance. 这样滥用药物会导致抗药性。
To develop effective combination of drug resistance modulators. 目的:寻找有效的多药耐药联合逆转剂。
Methods:The reversal effects of quinine(Quin) in combination with cyclosporin A (CsA),dipyridamole (DPM) or tamoxifen(Tam),respectively on the drug resistance of K562/HHT were studied by MTT,flow cytometry and median?effect principle. 方法：用MTT法、联合效应分析和流式细胞仪测定技术研究奎宁（Quin）分别与环孢菌素A（CsA）、他莫西酚（Tam）、潘生丁（DPM）联合逆转耐药细胞系K562/HHT对柔红霉素（DNR）和高三尖杉酯碱（HHT）耐药。
drug resistance reverse 耐药逆转
Drug resistance surveillance of pulmonary tuberculosis in Jiaxing City. 嘉兴市肺结核病耐药监测。
Study on infections and drug resistance of mycoplasma in female vaginalis. 株解脲支原体耐药分析。
Objective To study drug resistant mechanism and reversal of glioma we established C 6 MDR drug resistant cell line(C 6/adr). 目的为研究脑胶质瘤的耐药、机理及逆转 ,建立了C6耐阿霉素细胞株 (C6/adr)MDR细胞系。
And similar to penicillin, many bacteria also have had a sulfa drug resistance. 和青霉素类似，很多细菌也对磺胺产生了耐药性。
IFN combined with ATRA can partially revert drug resistance in MR2 cell. 2：IFN 联合ATRA能够部分逆转MR2细胞耐药。
Drug resistance is the main restriction factor for clinical usage of taxanes. 耐药是影响紫杉类药物临床疗效的重要因素。
Keywords chronic myelogenic leukemia;K562/MDR1;imatnib mesylate;arsenic trioxide;drug resistant reverse; 慢性粒细胞白血病;K562/MDR1;甲磺酸伊马替尼;三氧化二砷;逆转耐药;
If the bacteria glow, it means they are drug resistant. 如果细菌发光，这意味着它们具有耐药性。
The SPC-A1/Docetaxel cell line showed high resistance to docetaxel, and had the characteristic of multidrug resistance. The cell line could be useful for the study of drug resistant mechanism and its reversal of docetaxel. 经体外诱导耐药建立的SPC-A1/Docetaxel表现出对多西他赛较高的耐药性,并呈多药耐药特性,耐药细胞系SPC-A1/Docetaxel与亲本细胞SPC-A1的生物学特性有明显差异,可用于对多西他赛的耐药机制和逆转耐药研究。
reversal agent of drug resistance 耐药逆转剂
The tolerance of bone marrow to Ara C was remarkably increased after transfected by drug resistance gene. 耐药基因转染后小鼠骨髓对阿糖胞苷的耐受明显增加。
It seems likely that the drug resistance is caused by permeability changes rather than enzyme deficiency. 抗药性似乎是由渗透性改变而不是酶缺损而引起的。