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A study of tumor suppressor genes on chromosome 9 in glioblastomas. 胶质母细胞瘤9号染色体肿瘤抑制基因的研究。
The advances in the study of tumor suppressor genes in gliomas were reviewed. 作者就目前在脑胶质瘤中研究较多的抑癌基因的进展作了综述。
BRCA1 inactivates a tumor suppressor gene known as PTEN. BRCA1能够使人体内一种叫PTEN的肿瘤抑制基因失去活性。
In contrast, undifferentiated carcinomas rarely exhibit structurally altered tumor suppressor genes. 相反,未分化癌极少表现出抑癌基因结构的改变。
Objective: To study the expressions of pro-onc and tumor suppressor genes in gastric cancer by cDNA expression array. 目的:从基因水平了解正常胃和胃癌组织原癌基因和抑癌基因表达的分子机制。
The list of cancer-related mutations has grown to more than 100 oncogenes and 15 tumor suppressor genes. 与癌症有关的突变基因名单上，致癌基因已经增加到超过100个，而肿瘤抑制基因也有15个。
The expression of genes that inhibit cancer deelopment, so-called tumor suppressor genes, is often lost in tumor cells. 抑制癌症发生的基因即所谓的肿瘤抑制基因，其表达常在肿瘤细胞中缺失。
This silencing also affects the function of many tumor suppressor genes, which, in their unmethylated state, put the brakes on uncontrolled cell growth. 这种沉默也影响许多抑癌基因的功能，这些基因在非甲基化状态下可以阻止不受控制的细胞生长。
ON THE OTHER HAND, maybe cells can become malignant even before any master genes, oncogenes or tumor suppressor genes are mutated. 另一方面，或许在任何的主控基因、致癌基因或是肿瘤抑制基因产生突变之前，细胞就可能转变成恶性。
Now, the theory of oncogenes activity and tumor suppressor genes (TSG) inhibition can lead to cancer have been accepted. 目前，人们已经普遍接受了癌基因的激活和抑癌基因的失活可导致癌症发生的理论。
Until recently, many scientists believed that a tumor could take hold only after mutation had knocked tumor suppressor genes out of commission. 直到最近，许多科学家都还相信，当肿瘤抑制基因发生突变而受损之后，肿瘤才有机会立足；
The investigation on the role of DNA methylation in carcinogenesis have been mainly focused on promoter hypermethylation of tumor suppressor genes. 关于DNA甲基化在肿瘤中的作用的研究，大多集中在研究已知的抑癌基因启动子区的异常高甲基化。
CONCLUSION: RUNX3 gene may be one of the important candidate tumor suppressor genes in carcinogenesis of GC. 结论: RUNX3 可能是胃癌发生中重要的候选抑癌基因。
The investigations on the role of DNA methylation in carcinogenesis have been mainly focused on promoter hypermethylation of tumor suppressor genes. 关于DNA甲基化在肿瘤中的作用的研究，大多集中在研究已知的抑癌基因启动子区的异常高甲基化。
Like other tumors, development of ovarian cancer was related with the overexpression of oncogenes and the dysfunction of tumor suppressor genes. 卵巢癌的发生与其他肿瘤一样,存在癌基因的过表达与抑癌基因的失活。
The expression of genes can be “silenced” by hypermethylation of the tumor suppressor genes, which induces carcinogenesis. 抑癌基因启动子高甲基化可以使其表达失活,导致肿瘤发生。
Another worry is that the effect is temporary: methyl tags soon start popping up again, and the tumor suppressor genes switch back off. 另一项顾虑是药物可能只有暂时性的作用，甲基标签很快又会再度出现，肿瘤抑制基因就又遭到关闭。
Aberrant methylation of DNA is frequently found in tumor cells and tumor suppressor genes hypermethylation may also play an important role in carcinogenesis. 肿瘤可发生许多基因的异常甲基化,尤其是抑癌基因的高甲基化对肿瘤发生有很重要的作用。
The development of tumor involves in many factors, such as activating of oncogen, invalidating of cancer suppressor genes and abnormal expressing of apoptosis genes. 肿瘤的发生和发展涉及原癌基因的激活、抑癌基因的失活、凋亡相关基因的异常表达等,是涉及多基因、多因素的过程。
Although inactivation of some tumor suppressor genes such as p53 and p16INK4A has been identified, no known oncogene is commonly activated in hepatocellular carcinomas. 肝癌是亚洲及非洲最常见的恶性肿瘤，其原因可能是由于这些地区肝炎病毒的流行以及经常接触肝癌的特异性致癌物质。