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- GPC could promote the PKC protein expression and improve the proliferative activity of the mice hepatic cells. 葡多酚可促进小鼠肝细胞PKC的表达,提高肝细胞增殖活性。
- Conclusion:Eupolyphaga Seu Steleophaga could protect hepatic cells,and reduce the proliferation of fibrous tissue. C、LN、HA的作用,并能改善肝脏组织病理学变化。
- The patho- logical changes of hepatic cells in high dose group recovered to normal after 30 d of discontinuation of CSV. 其余动物于停药30天后处死,高剂量组肝细胞及肺泡正常。
- Objective:To examine whether VLDL or LDL can stimulate the secretion of plasminogen activator inhibitor type 1 (PAI-1) in hepatic cells. 目的 :观察极低密度脂蛋白 (VL DL)与低密度脂蛋白 (L DL)是否能刺激肝细胞纤溶酶原激活物抑制物 - 1(PAI- 1)的分泌。
- Conclusions The serum levels of hydroxide proline and hexosamine can signal hepatic cell damage induced by amatoxin. 结果:中、高剂量染毒组大鼠血清羟脯氨酸、氨基己糖含量显著低于对照组。
- Conclusion GPC can promote the PKC protein expression and improve the proliferative activity of the mice's hepatic cells. 结论葡多酚可促进小鼠肝细胞PKC表达,提高肝细胞增殖活性。
- Abstract ABSTRACT:Objective To research on the main pattern of hepatic cells death during hepatic ischemia/reperfusion(I/R) injury in cirrhotic rat. 摘要 摘要：目的 研究肝硬化大鼠肝缺血再灌注(I/R)损伤时肝细胞死亡的主要方式。
- Intrahepatic cholestasis results from the disturbance of the structure and function of bile secretory apparatus in hepatic cells. 肝内胆汁淤积是肝细胞内胆汁分泌器结构与功能严重障碍的结果,目前其发病机制尚不十分清楚。
- Objective To study the effect of grape procyanidins(GPC)on the protein kinase C(PKC) expression in mice's hepatic cells in viro. 目的体外观察葡多酚(GPC)对小鼠肝细胞蛋白激酶C(PKC)表达的影响。
- Conclusion FNH is a benign hepatocytic lesion, i.e. a reactive proliferation of hepatic cells to local blood vessel anomalies. 结论局灶性结节性增生是一种肝细胞对局部血管异常的反应性增生。
- More distruction of ultrastructural changes of hepatic cells in IR group was found than in DOX-IR group by transmission electron microscope. 透射电镜检查IR组肝细胞细可见明显的细胞器损害现象,DOX-IR细胞器受损较轻。
- In an early embryo, a bile canaliculus has been found.A bile duct is evolved from original hepatic cell. 胆小管在胚胎早期已经形成，胆管由肝母细胞演化而来；
- Conclusion GPC could promote the PKC protein expression and improve the proliferative activity of the mice hepatic cells. 结论葡多酚可促进小鼠肝细胞PKC的表达,提高肝细胞增殖活性。
- RESULTS: Severe damages of the hepatic cells of the N group including increased lysosomes and swollen and fused mitochondria were detected. 结果：N组肝细胞损害较严重，细胞内线粒体肿胀融合，溶酶体增多，肝血窦内疟原虫少见。
- Methods:Human hepatic cells BEL 7402 was cultured and different concentration of Antivirus Compound was added into the culture media with the control group. 方法 :采用 MTT(细胞毒 )实验培养人肝癌细胞株 BEL - 740 2细胞 ,加入不同浓度安体维康药液进行培养与对照组进行比较。
- Objective To study the effect of grape procyanidins (GPC) on the protein kinase C(PKC) and proliferative cell nuclear antigen (PCNA) expression in mice hepatic cells. 目的观察葡多酚(GPC)对小鼠肝细胞蛋白激酶C(PKC)和增殖细胞核抗原(PCNA)表达的影响。
- Objective To explore the biochemical indexes for hepatic cell damage induced by amatoxin in the early clinical diagnosis. 摘要 目的:探讨敏感的鹅膏毒肽致肝脏损伤的早期临床诊断生化指标。
- Compared to the control group, the necrotic area of hepatic lobule and fat droplets of hepatic cells were decreased and SDH activity was increased in the experimental group... 实验组与对照组相比,肝小叶损伤区域缩小,肝细胞中脂滴减少,琥珀酸脱氢酶活性增强。
- Conclusion Oncosis is the main pattern of hepatic cells death during I/R injury in cirrhotic rat, and the severity of hepatic injury correlates with the oncosis. 结论 胀亡是肝硬化大鼠肝脏I/R损伤肝细胞死亡的主要方式，肝功能损伤与胀亡密切相关。
- Conclusion Multiple risk factors affect cir-rhotic patients.The main death causes are liver function failure，hepatic cell carcinoma and GI bleeding. 结论肝硬化患者的病情严重程度和预后受到因素影响，死亡主要原因为肝功能衰竭、肝细胞癌、消化道出血。
