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- WI-38细胞株 WI-38 cells(CCL-75)
- 威斯塔研究院38细胞株 WI-38 cells; Wistar Institute 38 cells; Wistar's Insitute 38 cell
- 细胞 cell
- SWO-38细胞 SWO -38 cells
- 细胞株 cell strain
- 人胶质瘤SWO-38细胞 glioma
- 人淋巴因子激活的杀伤细胞对肝癌细胞株凋谢作用的观察 A Observaion of Apoptoisi of Liver Cancer Cells Induced by Human LAK Cells
- 人参皂苷Rg1可能通过激活端粒酶活性和减少端粒长度缩短而发挥其抗t BHP诱导的WI 38细胞衰老作用。 Activation of telomerase and prolonging of RTF length might be involved in t he process of ginsenoside Rg1 protection against t-BHP-induced senescence in W I-38 cells.
- T24细胞株 T24 Cell lines
- 然后，将WI-38细胞随机分为4组，用不同剂量人参皂甙Rg1预处理，观察其对t-BHP诱导的细胞衰老的影响。 After four stresses of t-BHP, we compared cells in three aspects above.
- NB4细胞株 NB4 cell line
- 1 .t一BHP可诱导Wl一38细胞衰老，可用于建立体外细胞衰老模型。 Conclusion: t-BHP can be used to induce WI-38 cells senescence and to establish a model of premature senescence.
- 肝细胞株 hepatocyte lines
- Rgl预处理可明显减弱t-BHP对WI-38细胞衰老的诱导作用,同时p21表达水平明显降低,cyclin E和CDK2表达水平增加。 Pretreatment with Rgl significantly attenuated t-BHP-induced senescence in WI-38 cells. Simultaneously, compared with cells treated with t-BHP alone, Rgl pretreatment markedly decreased the level of p21 protein and increased the levels of CDK2 and cyclin E.
- A20细胞株 A20 cell line
- B9细胞株 B9 cell line
- 方法 :将WI 38细胞随机分为 4组 ,用不同剂量Rg1预处理。 从 30代开始 ,隔代用t BHP作用 ,每次 1h ,共 4次 ,诱导细胞衰老。 METHODS: WI 38 cells were divided into 4 groups and randomly added with different concentrations of Rg1. From 30 population doubling (PD), WI 38 cells were exposed to t BHP for 1 h at every two PDs.
- CEM细胞株 CEM cell line
- CHO细胞株 CHO cell
- CNE细胞株 CNE
