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- TFAR-19凋亡相关蛋白 TFAR-19 apoptosis related protein
- 大田软海绵酸对FL细胞DNA的损伤及凋亡相关蛋白表达的影响 DNA DAMAGE AND ALTERATION OF EXPRESSION IN APOPTOSIS RELATED PROTEINS INDUCED BY OKADAIC ACID IN FL CELLS
- 石斛合剂对衰老糖尿病大鼠胰腺组织凋亡相关基因Bax,Bcl-2mRNA及蛋白表达的调控 The regulation of Dendrobium Compound (DC) on mRNA and protein expression of Bax, Bcl-2 genes associated with apoptosis in aged diabetic rats' pancreas
- 凋亡相关蛋白 Apoptosis-associated proteins
- 凋亡相关蛋白基因 Apoptosis - related protein
- rhGM-CSF/IL-3融合蛋白对Ara-C介导的HL-60细胞凋亡的影响 The Effect of rhGM-CSF/IL-3 Fusion Protein on Apoptosis of HL-60 Cells Mediated by Ara-C
- 细胞凋亡及凋亡相关基因在下肢静脉曲张中的表达及意义 The expression and significance of cell apoptosis and apoptosis genes of varicosity in the lower extremities
- 右-苯丙胺对脑缺血大鼠细胞凋亡和生长相关蛋白43的作用 Effect of D-amphetamine on apoptosis and GAP-43 expression of rat brain cells after focal cerebral ischemia
- 用定量RT-PCR方法检测脑梗死半影区内凋亡相关基因的表达 Quantitative RT-PCR in detecting the apoptosis associated gene expression in penumbra area of cerebral infarction in rats
- 测定凋亡相关抗原7A6表达率可预测急性粒细胞白血病疗效。 In result,determination of expressing rate of antigen 7A6could predict efficacy of chemotherapy in AML.
- 凋亡相关基因 apoptosis related gene
- 凋亡相关结构域 caspase recruitment domain
- 黄苔胃病患者胃粘膜凋亡基因及相关蛋白的表达 Study on Apoptosis Gene and its Related Protein in Gastric Mucosa in Gastric Disease Patients with Yellow Tongue Coating
- 细胞凋亡相关基因 Apoptosis-related genes
- 细胞凋亡相关基因Fas和FasL在大鼠苯巴比妥促肝癌过程中的表达研究 Fas and FasL expressions in rat liver tumor promotion by phenobarbital
- 慢性胃炎脾胃湿热证患者胃粘膜细胞凋亡及相关蛋白的研究 Study on Apoptosis Index and Its Gene Related Protein in Gastric Mucosa of Patients with Chronic Gastritis of Spleen-Stomach Damp-Heat Syndrome Type
- 辛伐他汀诱导大鼠血管平滑肌细胞凋亡及对凋亡相关基因表达的影响 Simvastatin induced apoptosis and its effect on apoptosis-related gene expression in rat vascular smooth muscle cell
- 凋亡相关基因/分析 apoptosis genes/analysis
- 凋亡相关基因或因子 genes factors
- 肿瘤坏死因子相关凋亡诱导配体通过促进死亡相关蛋白3表达抑制人胃癌细胞株的生长 Tumor necrosis factor-related apoptosis-induced ligand inhibits the growth of human gastric cancer cell lines via enhancing death-associated protein 3 expression