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- PI3K/AKT PI3K/AKT
- PI3K-Akt通路 PI3K - Akt signal pathway
- PI3K/Akt/mTOR PI3K/Akt/mTOR signal pathway
- PI3k/Akt抑制剂 PI3k/Akt inhibitor
- PI3K-AKT信号通路 PI3K-AKT signal pathway
- PI3K PI3 K
- p-AKT p-AKT
- 基因Akt genes Akt
- Wortmannin抑制PI3K途径对K562细胞增殖和Ara-C诱导的细胞凋亡的影响 Effects of PI3K on K562 Cell Proliferation and Apoptosis Induced by Ara-C
- AKT蛋白 AKT protein
- Akt通路 Akt signal pathway
- 利用信号通路特异性抑制剂抑制实验，提示PI3K在ADAS细胞向内皮细胞分化中 And PI3 kinase inhibitor LY294002 blocked the differentiation of ADAS cells into endothelial cells in vitro.
- PI3K-Akt PI3K-Akt
- PI3K／Akt PI3K/Akt
- 原癌基因产物c CBL对BCR/ABL酪氨酸激酶活性无反向调节作用 ,它是连接BCR/ABL与PI3K之间的重要桥梁分子。 c CBL Has no regulatory effects on BCR/ABL tyrosine kinase. c CBL is assumed to be the linkage between BCR/ABL and PI3K and subsequently activates PI3K. [
- PKB／Akt PKB/Akt
- Akt／PKB Akt kinase/protein kinase B
- Akt／mTOR Akt/mTOR
- 作用 2 4h后细胞实验组细胞BCL -2蛋白表达较对照组降低 ,说明Wortmannin可促进K5 62细胞的凋亡。 结论 Wortmannin可以通过抑制PI3K通路抑制K5 62细胞的增殖。 Conclusion Wortmannin could inhibitedtheproliferationofK5 6 2cells ,andwascellcyclespecificagent (CCSA) .
- P13K／Akt PI3 K/Akt
