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- perivascular gliosis 血管周围神经胶质增生
- Gliosis and gliocyte apoposis were also observed. 所有病例可见胶质细胞增生和凋亡。
- Perivascular T lymphocytes are mainly auxiliary. 血管周围的淋巴细胞主要是T辅助。
- Gliosis around motor neurons was seen. 神经元周围胶质细胞增生。
- Since gliomas donot give this perivascular cuff feature. 因为胶质瘤不会有血管周围套特征。
- A gliosis layer between the wall of tumor and the hypothalamus was seen. 肿瘤与下丘脑间有胶质增生层。
- Did you see perivascular inflammation or eosinophilic granular bodies? 你们看到血管周炎症或嗜酸颗粒小体了吗?
- The vascular changes interfere with diffusion of nutrients from plasma into adjacent perivascular tissue and cells. 这些血管变化干扰了营养物质从血浆中向血管周围邻接的组织和细胞扩散。
- The signal characteristics probably result from dense gliosis and diminished myelin sheaths in the hemartomatous areas. 病变的信号特点很可能是因为错构瘤区域致密的胶质增生和髓鞘都是造成的。
- Production of chemokines by perivascular adipose tissue: a role in the pathogenesis of atherosclerosis? 血管周围脂肪组织趋化产物:在动脉硬化发病机理中的作用?
- Objective To discuss the etiology, chinical characteristics, pathology and canceration in gliosis. 目的分析讨论脑胶质细胞增生症的病因、临床特点、病理及其演变。
- Conclusion EAE is characterised by perivascular inflammatory cell infiltration and demyelination in white matter. 结论 EAE的病理改变主要为血管周围炎性浸润及白质脱髓鞘。
- Conclusion Gliosis is a nonmalignant disease, however, it has the possibility of canceration. 结论脑胶质呼吸细胞增生是良性病性病理改变,但有恶变的报道可能,应进行定期随访。
- These are normal, and represent perivascular spaces of the lenticulostriate arteries. 这是正常的纹状体动脉周围的血管区域。
- Method 34 cases of gliosis were analyzed retrospectively follow-up for a long period. 方法对34例脑胶质细胞增生症病例进行回顾性研究、通过长期随访和病理切片复查进行分析。
- White matter demyelination, neural axis compression deformation, gliosis and nerve cell degeneration and so on. 白质脱髓鞘变,神经轴受压变形,胶质增生及神经细胞退行性变等。
- The pinealocytic processes terminated in the perivascular connective tissue space. 松果体细胞的突起终止于血管周隙内;
- Dermal papilla layer of capillary proliferation and expansion of perivascular infiltration of lymphocytes dominated. 真皮乳头层内毛细血管增生和扩张,血管周围有以淋巴细胞为主的细胞浸润。
- When VEGF activity is impaired, tumor cells invade deep into the brain in the perivascular compartment. 当VEGF活性受到损坏,肿瘤细胞侵入大脑血管管腔的周围。
- PML lesions have myelin loss, associated macrophages, even the perivascular inflammation. PML有髓鞘丢失、相关的巨噬细胞以及血管周围炎症细胞。