SDS-PAGE and IEF analyses showed that the mutants without PI6.82 polypeptide in common while forming / increasing other polypeptide indicating that the mutants were controlled by structural genes.

• SDS-PAGE和IEF分析表明这些突变体在共同缺失1条pI6。82多肽的同时，或形成新的多肽，或其他多肽表现量增加，这些突变体是由结构基因控制的。