Inhaled nitric oxide downregulates iNOS immunoreactivity and has no effect on the expression of eNOS, which attentuates the reperfused lung injury.

• 1再灌注肺组织 e NOS表达减弱 ；i NOS表达增强 ；导致 NO合成增多 ；造成肺组织缺血灌注损伤 .；2吸入NO能抑制肺组织 i NOS表达；维持 e NOS在内皮细胞的正常表达；减轻肺再灌注损伤