In HCV-infected liver the combination of enhanced mDC function and a reduced number of pDCs may contribute to viral persistence in the face of persistent inflammation.

• 在HCV感染的肝脏中，增强的髓样树状突细胞功能和降低的浆细胞样树状突细胞的数量的联合可能促成在持久炎症的面前的病毒持续感染。