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- Methods Focal brain ischemia animal model was used. 方法应用局限性脑缺血动物模型。
- Disturbances of calcium homeostasis and modification of ca~(2+) pump(Ca~(22+),Mg~(2+)-ATPase)in the rabbit focal brain ischemia. 脑缺血时钙稳态与钙泵(Ca~(2+),Mg~(2+)-ATP酶)活性变化
- Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
- Effects of mild hypothermia on heart of rats with focal brain ischemia 亚低温治疗实验性脑梗死时对心脏的影响
- Reduced infarction area of exogenous basic fibroblast growth factor in a rat focal brain ischemia model 外源性碱性成纤维细胞生长因子对大鼠局灶性脑缺血的影响
- Effects of the systemic mild hypothermia on heart of rats with focal brain ischeMia 亚低温治疗大鼠局灶性脑缺血时对心脏的影响
- EFFECTS OF GINSENOSIDES ON THE Ach AND ChAT IN EXPERIMENTAL MODELS OF FOCAL BRAIN ISCHEMIA 人参皂甙对实验性脑缺血脑组织内Ach、ChAT的影响
- Focal brain ischemia 局灶性脑缺血
- CDP-choline improved the recoveryof SEPs caused by brain ischemia obviously. CDP-choline可明显促进缺血脑功能的恢复。
- CONCLUSION: UCB-MSCs effectively traverse blood-brain barrier, and migrate, disperse and conform around focal brain tissues. 结论：脐血间充质干细胞能有效透过血脑屏障，在病灶脑组织周围迁移、扩散、整合；
- But the detailed mechanisms of G-LOC or G induced brain ischemia as well as protective effect IPC were not clear. 然而不论大G昏迷、G力造成的脑损伤及缺血前制约效应的保护机转都并不清楚。
- It can cause severe complications, such as brain ischemia or cervical hematorrhea incurred by rupture of aneurysm. 它可以引起严重的并发症，比如大脑局部缺血或动脉瘤破裂产生的颈部软组织出血。
- It was suggested that DDPH had some protective effect on acute brain ischemia and ischemia-reperfusion. 结果提示，DDPH对动物脑缺血具有一定的保护作用。
- Objective To study the improvement effects of GM_1 on brain ischemia reperfusion(BIR) induced memory impairment in mice. 目的探讨脑缺血复灌后连续应用GM1对改善记忆障碍的疗效。
- Without obvious influence in brain metabolic functions,L1 KO mice is an useful animal model for investigation of the role of L1 in brain ischemia. L1CAM基因缺失对小鼠脑局部代谢功能无明显影响,提示L1 KO小鼠可作为研究L1CAM在脑缺血中作用的动物模型。
- The abnormal DA release inhibited by L-NNA during global ischemia may be one of the protective mechanism of this agent on brain ischemia. L-NNA抑制半球缺血时DA的不正常释放可能是其保护缺血大脑的机制之一。
- Keywords The kidney injury after brain ischemia reperfusion was correlated with the dysequilibrium between ET and CGRP in which ET was dominant. 脑缺血/中医疗法;益元活血丹/治疗应用;大黄/治疗应用;血栓心脉宁/治疗应用;再灌注损伤;肾疾病/中医药疗法;肾疾病/病理学;肾疾病/超微结构;内皮缩血管肽类/代谢;
- When the collateral circulation is good, the radialis velocity may be normal, Whereas, the risk of brain ischemia would be increase. 反之可能会诱发脑缺血的临床症状。
- Chan ges of bcl-xl protein expression were observed at 24 hours after global brain ischemia, and apoptosis were checke d at 72 hours in hippocampal CA1 subregion. 观察缺血后 2 4h海马CA1区bcl -xl蛋白表达和缺血后 72h细胞凋亡。
- GU WP, YANG QD, XIE GJ.Experimental delayed neure death present apoptosis after brain ischemia refilling [J].China Journal of Modern Medicine, 2001, 11(3): 30-31. [2]谷文萍;杨期东;谢光洁.;实验性脑缺血再灌后迟发性神经元死亡表现细胞凋亡[J]
