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- Effects of Congsheng Capsule on cultured rat cerebral cortical neurons after ischemia-reperfusion. 聪圣胶囊对拟缺血再灌注大鼠原代培养皮层神经细胞的保护作用。
- CEP 11004 inhibited the activation of SAPK/JNK to protect cortical neurons from apoptosis induced by aluminum chloride. CEP 110 0 4可能通过抑制SAPK/JNK的活性 ,对AlCl3诱导的皮层神经元凋亡产生保护作用
- RhoA was also located in the lipid rafts of brain and cerebellar granule cells as well as cortical neurons. RhoA 也可分布于成年大鼠大脑、小脑颗粒细胞以及皮质神经元的Lipid Rafts 上。
- This technique for the primary culture of cortical neurons is an ideal good technique for the general labs. 本方法适合普通实验室进行大鼠皮层神经元体外培养。
- It is an ideal technique for culture of cortical neurons from cerebral cortex of neonate rats in vitro. 结论：该培养技术是大鼠皮层神经元体外培养的一种较理想的方法。
- Amyloid-beta at sublethal level impairs BDNF-induced arc expression in cortical neurons. - 中国生物学文摘 - Wang DC Chen SS Lee YC Chen TJ.
- Title： Effects of Congsheng Capsule on cultured rat cerebral cortical neurons after ischemia-reperfusion. 关键词：聪圣胶囊;神经细胞;脑缺血再灌注;细胞凋亡
- Electrical activity in the form of nerve impulses being sent and received to and from cortical neurons is always present, even during sleep. 大脑皮层神经元不停地发送、接收神经冲动信号，即使在睡觉时也不例外。
- AIM: To explore the possible mechanism of tert - butyl hydroperoxide (t - BHP) - induced apoptosis in rat cortical neurons. 目的:探讨第三丁基过氧化氢(t-BHP)诱导大鼠皮层神经元凋亡的可能机制。
- They suggest that repeated firing of hyperexcitable cortical neurons may allow extracellular potassium accumulation which initiates SCD. 他们提出，高兴奋性皮质区域神经元的重覆放电可能会使细胞外钾离子蓄积，因而触发了散布的皮质忧郁症。
- Objection To investigate the effect of EGB761 on cultured cortical neurons after oxygen and glucose deprivation （OGD）and reoxygenation injury. 目的 研究银杏叶提取物（EGB761）对体外培养神经元缺糖缺氧/复糖复氧性损伤的保护作用，并初步探索其可能的作用机制。方法 利用体外培养的皮层神经元，通过去除培养液中的糖和氧（oxygen and glucose deprivation，OGD）模拟缺血缺氧，恢复糖氧供给模拟再灌注。
- Through the mechanism called locally excitatory globally inhibitory existing among the visual cortical neurons,neurons can oscillate in terms of visual information. 视觉皮层神经元之间存在的局部兴奋全局抑制机制能够使神经元根据接受到的视觉信息产生相应的振荡活动。
- Newborn rat cerebral cortical neurons were used as a culture model to test the neuronotrophic factors (NTFs) and neuritepromoting factors (NPFs) in EWTE. 并建立新生大鼠大脑皮质神经元体外培养模型,检测BWTE中的神经元营养因子(NTFs)和促神经突起生长因子(NPFs)。
- The effects of ?l-3-n-butylphthalide(?l-NBP) and ?d-3-n-butylphthalide(?d-NBP) on hypoxia/hypoglycemia?induced cytotoxicity in primary cultured rat cortical neurons were studied. 为进一步探讨丁基苯肽对神经的保护作用，用原代培养大鼠皮层神经细胞的方法，以LDH和细胞形态等指标，观察了l?丁基苯酞（l?NBP）和d?丁基苯酞（d?NBP）对低糖低氧诱导的大鼠皮层神经细胞损伤的保护作用。
- The component of the extracellular matrix called the perineuronal net (PN), as its name implies, surrounds the cell body and proximal dendrites of selected cortical neurons. 细胞外基质的成分是围神经元网络组织，顾名思义它位于特定皮质区的神经元胞体和近段树突周围。
- Conclusion Thiopental sodium can increase the excitation threshold of brain cortical neurons and decrease the level of convulsive seizure induced by ECT. 结论硫喷妥钠可上调大脑皮质神经元的兴奋阈值，降低电抽搐治疗时的痉挛发作程度。
- In itro results showed that leptin, 50 to 100 microg/mL, protected primary cortical neurons against death induced by oxygen-glucose depriation in a concentration-dependent manner. 体外实验结果显示50-100mg/ml的来普汀可以浓度依赖性的保护原代皮层神经元免受由缺氧缺糖所导致的细胞凋亡。
- Wang KW, Yang ZH, Wang ZG, et al.Expression of C-fos proteinafter cultured cortical neuron injury in rats [J].Chin J Traumatol, 1998, 14(4): 219-22. [2]王克万;尹志勇;杨志焕;等.;培养大鼠皮层神经元损伤后c-fos蛋白表达[J]
- Methods: (1)Embryonic cerebral cortical neurons of a rat were cultured in vitro by combined method of typspin digestion and DNase I based on the neurobasal medium. 第一部分：大鼠胚脑皮质神经元细胞的原代培养、鉴定和缺氧模型的建立目的：进行大鼠胚脑皮质神经元细胞培养和鉴定，并建立大鼠胚脑皮质神经元细胞培养的缺氧模型。
- Methods The seizure model was recurrent epileptiform discharges induced by transient magnesium-free media treatment in the primary cultured embryo cortical neurons of rat. 方法 以原代培养的大鼠大脑皮层神经元无镁诱导的反复惊厥样放电为模型。
