At the cellular level, bisphosphonates inhibit local osteoclast recruitment to bone surfaces and reduce the ability of osteoclasts to bind to bone, thereby interfering with osteoclast function.

• 在分子水平上，双磷酸盐阻止了局部的破骨细胞在骨表面的聚集并且减低了破骨细胞依附于骨的能力，因此干涉了破骨细胞功能的实现。